node1 | node2 | node1 accession | node2 accession | node1 annotation | node2 annotation | score |
KMT65171.1 | KMT65412.1 | XM47_10565 | XM47_08610 | Recombination and repair protein; May be involved in recombinational repair of damaged DNA. | DNA polymerase V; Derived by automated computational analysis using gene prediction method: Protein Homology; Belongs to the peptidase S24 family. | 0.629 |
KMT65171.1 | KMT66150.1 | XM47_10565 | XM47_05100 | Recombination and repair protein; May be involved in recombinational repair of damaged DNA. | Hypothetical protein; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.578 |
KMT65171.1 | KMT66843.1 | XM47_10565 | XM47_01640 | Recombination and repair protein; May be involved in recombinational repair of damaged DNA. | Hypothetical protein; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | 0.803 |
KMT65171.1 | lexA | XM47_10565 | XM47_14490 | Recombination and repair protein; May be involved in recombinational repair of damaged DNA. | LexA family transcriptional regulator; Represses a number of genes involved in the response to DNA damage (SOS response), including recA and lexA. In the presence of single-stranded DNA, RecA interacts with LexA causing an autocatalytic cleavage which disrupts the DNA-binding part of LexA, leading to derepression of the SOS regulon and eventually DNA repair. | 0.629 |
KMT65171.1 | mfd | XM47_10565 | XM47_12030 | Recombination and repair protein; May be involved in recombinational repair of damaged DNA. | Transcription-repair coupling factor; Couples transcription and DNA repair by recognizing RNA polymerase (RNAP) stalled at DNA lesions. Mediates ATP-dependent release of RNAP and its truncated transcript from the DNA, and recruitment of nucleotide excision repair machinery to the damaged site; In the C-terminal section; belongs to the helicase family. RecG subfamily. | 0.648 |
KMT65171.1 | ppnK | XM47_10565 | XM47_09810 | Recombination and repair protein; May be involved in recombinational repair of damaged DNA. | Inorganic polyphosphate kinase; Involved in the regulation of the intracellular balance of NAD and NADP, and is a key enzyme in the biosynthesis of NADP. Catalyzes specifically the phosphorylation on 2'-hydroxyl of the adenosine moiety of NAD to yield NADP. | 0.862 |
KMT65171.1 | recA | XM47_10565 | XM47_04755 | Recombination and repair protein; May be involved in recombinational repair of damaged DNA. | Recombinase RecA; Can catalyze the hydrolysis of ATP in the presence of single- stranded DNA, the ATP-dependent uptake of single-stranded DNA by duplex DNA, and the ATP-dependent hybridization of homologous single-stranded DNAs. It interacts with LexA causing its activation and leading to its autocatalytic cleavage; Belongs to the RecA family. | 0.833 |
KMT65171.1 | ruvA | XM47_10565 | XM47_08960 | Recombination and repair protein; May be involved in recombinational repair of damaged DNA. | ATP-dependent DNA helicase RuvA; The RuvA-RuvB complex in the presence of ATP renatures cruciform structure in supercoiled DNA with palindromic sequence, indicating that it may promote strand exchange reactions in homologous recombination. RuvAB is a helicase that mediates the Holliday junction migration by localized denaturation and reannealing. RuvA stimulates, in the presence of DNA, the weak ATPase activity of RuvB. | 0.615 |
KMT65171.1 | uvrC | XM47_10565 | XM47_16965 | Recombination and repair protein; May be involved in recombinational repair of damaged DNA. | Excinuclease ABC subunit C; The UvrABC repair system catalyzes the recognition and processing of DNA lesions. UvrC both incises the 5' and 3' sides of the lesion. The N-terminal half is responsible for the 3' incision and the C-terminal half is responsible for the 5' incision. | 0.578 |
KMT65171.1 | xseA | XM47_10565 | XM47_10295 | Recombination and repair protein; May be involved in recombinational repair of damaged DNA. | Hypothetical protein; Bidirectionally degrades single-stranded DNA into large acid- insoluble oligonucleotides, which are then degraded further into small acid-soluble oligonucleotides; Belongs to the XseA family. | 0.667 |
KMT65412.1 | KMT65171.1 | XM47_08610 | XM47_10565 | DNA polymerase V; Derived by automated computational analysis using gene prediction method: Protein Homology; Belongs to the peptidase S24 family. | Recombination and repair protein; May be involved in recombinational repair of damaged DNA. | 0.629 |
KMT65412.1 | KMT66150.1 | XM47_08610 | XM47_05100 | DNA polymerase V; Derived by automated computational analysis using gene prediction method: Protein Homology; Belongs to the peptidase S24 family. | Hypothetical protein; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.624 |
KMT65412.1 | KMT66843.1 | XM47_08610 | XM47_01640 | DNA polymerase V; Derived by automated computational analysis using gene prediction method: Protein Homology; Belongs to the peptidase S24 family. | Hypothetical protein; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | 0.627 |
KMT65412.1 | recA | XM47_08610 | XM47_04755 | DNA polymerase V; Derived by automated computational analysis using gene prediction method: Protein Homology; Belongs to the peptidase S24 family. | Recombinase RecA; Can catalyze the hydrolysis of ATP in the presence of single- stranded DNA, the ATP-dependent uptake of single-stranded DNA by duplex DNA, and the ATP-dependent hybridization of homologous single-stranded DNAs. It interacts with LexA causing its activation and leading to its autocatalytic cleavage; Belongs to the RecA family. | 0.972 |
KMT66150.1 | KMT65171.1 | XM47_05100 | XM47_10565 | Hypothetical protein; Derived by automated computational analysis using gene prediction method: Protein Homology. | Recombination and repair protein; May be involved in recombinational repair of damaged DNA. | 0.578 |
KMT66150.1 | KMT65412.1 | XM47_05100 | XM47_08610 | Hypothetical protein; Derived by automated computational analysis using gene prediction method: Protein Homology. | DNA polymerase V; Derived by automated computational analysis using gene prediction method: Protein Homology; Belongs to the peptidase S24 family. | 0.624 |
KMT66150.1 | KMT66843.1 | XM47_05100 | XM47_01640 | Hypothetical protein; Derived by automated computational analysis using gene prediction method: Protein Homology. | Hypothetical protein; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | 0.774 |
KMT66150.1 | lexA | XM47_05100 | XM47_14490 | Hypothetical protein; Derived by automated computational analysis using gene prediction method: Protein Homology. | LexA family transcriptional regulator; Represses a number of genes involved in the response to DNA damage (SOS response), including recA and lexA. In the presence of single-stranded DNA, RecA interacts with LexA causing an autocatalytic cleavage which disrupts the DNA-binding part of LexA, leading to derepression of the SOS regulon and eventually DNA repair. | 0.624 |
KMT66150.1 | recA | XM47_05100 | XM47_04755 | Hypothetical protein; Derived by automated computational analysis using gene prediction method: Protein Homology. | Recombinase RecA; Can catalyze the hydrolysis of ATP in the presence of single- stranded DNA, the ATP-dependent uptake of single-stranded DNA by duplex DNA, and the ATP-dependent hybridization of homologous single-stranded DNAs. It interacts with LexA causing its activation and leading to its autocatalytic cleavage; Belongs to the RecA family. | 0.540 |
KMT66150.1 | ruvA | XM47_05100 | XM47_08960 | Hypothetical protein; Derived by automated computational analysis using gene prediction method: Protein Homology. | ATP-dependent DNA helicase RuvA; The RuvA-RuvB complex in the presence of ATP renatures cruciform structure in supercoiled DNA with palindromic sequence, indicating that it may promote strand exchange reactions in homologous recombination. RuvAB is a helicase that mediates the Holliday junction migration by localized denaturation and reannealing. RuvA stimulates, in the presence of DNA, the weak ATPase activity of RuvB. | 0.405 |