| node1 | node2 | node1 accession | node2 accession | node1 annotation | node2 annotation | score |
| C3138 | PBPRA1578 | PBPRA0694 | PBPRA1578 | Putative DNA repair protein RecN; May be involved in recombinational repair of damaged DNA. | Hypothetical SulA, SOS-response cell division inhibitor, blocks FtsZring formation; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | 0.873 |
| C3138 | RECA | PBPRA0694 | PBPRA3068 | Putative DNA repair protein RecN; May be involved in recombinational repair of damaged DNA. | Putative RecA protein; Can catalyze the hydrolysis of ATP in the presence of single- stranded DNA, the ATP-dependent uptake of single-stranded DNA by duplex DNA, and the ATP-dependent hybridization of homologous single-stranded DNAs. It interacts with LexA causing its activation and leading to its autocatalytic cleavage; Belongs to the RecA family. | 0.903 |
| C3138 | S0300 | PBPRA0694 | PBPRA0832 | Putative DNA repair protein RecN; May be involved in recombinational repair of damaged DNA. | Putative DNA-damage-inducible protein P; Poorly processive, error-prone DNA polymerase involved in untargeted mutagenesis. Copies undamaged DNA at stalled replication forks, which arise in vivo from mismatched or misaligned primer ends. These misaligned primers can be extended by PolIV. Exhibits no 3'-5' exonuclease (proofreading) activity. May be involved in translesional synthesis, in conjunction with the beta clamp from PolIII. | 0.477 |
| C3138 | STY2088 | PBPRA0694 | PBPRA3003 | Putative DNA repair protein RecN; May be involved in recombinational repair of damaged DNA. | Putative DNA damage-inducible gene in SOSregulon, dependent on cyclic AMP and H-NS; Predicted by orpheus program; predicted by glimmer program. | 0.639 |
| C3138 | T4291 | PBPRA0694 | PBPRB0972 | Putative DNA repair protein RecN; May be involved in recombinational repair of damaged DNA. | Hypothetical protein involved in DNA repair; Predicted by orpheus program; predicted by glimmer program. | 0.477 |
| C3138 | UMUD | PBPRA0694 | PBPRB0973 | Putative DNA repair protein RecN; May be involved in recombinational repair of damaged DNA. | Hypothetical protein; Predicted by orpheus program; predicted by glimmer program; Belongs to the peptidase S24 family. | 0.749 |
| PBPRA1578 | C3138 | PBPRA1578 | PBPRA0694 | Hypothetical SulA, SOS-response cell division inhibitor, blocks FtsZring formation; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | Putative DNA repair protein RecN; May be involved in recombinational repair of damaged DNA. | 0.873 |
| PBPRA1578 | PBPRB1030 | PBPRA1578 | PBPRB1030 | Hypothetical SulA, SOS-response cell division inhibitor, blocks FtsZring formation; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | Hypothetical protein; Predicted by orpheus program; predicted by glimmer program. | 0.859 |
| PBPRA1578 | RECA | PBPRA1578 | PBPRA3068 | Hypothetical SulA, SOS-response cell division inhibitor, blocks FtsZring formation; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | Putative RecA protein; Can catalyze the hydrolysis of ATP in the presence of single- stranded DNA, the ATP-dependent uptake of single-stranded DNA by duplex DNA, and the ATP-dependent hybridization of homologous single-stranded DNAs. It interacts with LexA causing its activation and leading to its autocatalytic cleavage; Belongs to the RecA family. | 0.665 |
| PBPRA1578 | STY2088 | PBPRA1578 | PBPRA3003 | Hypothetical SulA, SOS-response cell division inhibitor, blocks FtsZring formation; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | Putative DNA damage-inducible gene in SOSregulon, dependent on cyclic AMP and H-NS; Predicted by orpheus program; predicted by glimmer program. | 0.861 |
| PBPRA1578 | UMUD | PBPRA1578 | PBPRB0973 | Hypothetical SulA, SOS-response cell division inhibitor, blocks FtsZring formation; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | Hypothetical protein; Predicted by orpheus program; predicted by glimmer program; Belongs to the peptidase S24 family. | 0.687 |
| PBPRB1030 | PBPRA1578 | PBPRB1030 | PBPRA1578 | Hypothetical protein; Predicted by orpheus program; predicted by glimmer program. | Hypothetical SulA, SOS-response cell division inhibitor, blocks FtsZring formation; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | 0.859 |
| PBPRB1030 | RECA | PBPRB1030 | PBPRA3068 | Hypothetical protein; Predicted by orpheus program; predicted by glimmer program. | Putative RecA protein; Can catalyze the hydrolysis of ATP in the presence of single- stranded DNA, the ATP-dependent uptake of single-stranded DNA by duplex DNA, and the ATP-dependent hybridization of homologous single-stranded DNAs. It interacts with LexA causing its activation and leading to its autocatalytic cleavage; Belongs to the RecA family. | 0.799 |
| PBPRB1030 | STY2088 | PBPRB1030 | PBPRA3003 | Hypothetical protein; Predicted by orpheus program; predicted by glimmer program. | Putative DNA damage-inducible gene in SOSregulon, dependent on cyclic AMP and H-NS; Predicted by orpheus program; predicted by glimmer program. | 0.495 |
| PBPRB1030 | UMUD | PBPRB1030 | PBPRB0973 | Hypothetical protein; Predicted by orpheus program; predicted by glimmer program. | Hypothetical protein; Predicted by orpheus program; predicted by glimmer program; Belongs to the peptidase S24 family. | 0.565 |
| RECA | C3138 | PBPRA3068 | PBPRA0694 | Putative RecA protein; Can catalyze the hydrolysis of ATP in the presence of single- stranded DNA, the ATP-dependent uptake of single-stranded DNA by duplex DNA, and the ATP-dependent hybridization of homologous single-stranded DNAs. It interacts with LexA causing its activation and leading to its autocatalytic cleavage; Belongs to the RecA family. | Putative DNA repair protein RecN; May be involved in recombinational repair of damaged DNA. | 0.903 |
| RECA | PBPRA1578 | PBPRA3068 | PBPRA1578 | Putative RecA protein; Can catalyze the hydrolysis of ATP in the presence of single- stranded DNA, the ATP-dependent uptake of single-stranded DNA by duplex DNA, and the ATP-dependent hybridization of homologous single-stranded DNAs. It interacts with LexA causing its activation and leading to its autocatalytic cleavage; Belongs to the RecA family. | Hypothetical SulA, SOS-response cell division inhibitor, blocks FtsZring formation; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | 0.665 |
| RECA | PBPRB1030 | PBPRA3068 | PBPRB1030 | Putative RecA protein; Can catalyze the hydrolysis of ATP in the presence of single- stranded DNA, the ATP-dependent uptake of single-stranded DNA by duplex DNA, and the ATP-dependent hybridization of homologous single-stranded DNAs. It interacts with LexA causing its activation and leading to its autocatalytic cleavage; Belongs to the RecA family. | Hypothetical protein; Predicted by orpheus program; predicted by glimmer program. | 0.799 |
| RECA | RPON | PBPRA3068 | PBPRA3255 | Putative RecA protein; Can catalyze the hydrolysis of ATP in the presence of single- stranded DNA, the ATP-dependent uptake of single-stranded DNA by duplex DNA, and the ATP-dependent hybridization of homologous single-stranded DNAs. It interacts with LexA causing its activation and leading to its autocatalytic cleavage; Belongs to the RecA family. | Putative sigma factor N RpoN; Sigma factors are initiation factors that promote the attachment of RNA polymerase to specific initiation sites and are then released. | 0.500 |
| RECA | S0300 | PBPRA3068 | PBPRA0832 | Putative RecA protein; Can catalyze the hydrolysis of ATP in the presence of single- stranded DNA, the ATP-dependent uptake of single-stranded DNA by duplex DNA, and the ATP-dependent hybridization of homologous single-stranded DNAs. It interacts with LexA causing its activation and leading to its autocatalytic cleavage; Belongs to the RecA family. | Putative DNA-damage-inducible protein P; Poorly processive, error-prone DNA polymerase involved in untargeted mutagenesis. Copies undamaged DNA at stalled replication forks, which arise in vivo from mismatched or misaligned primer ends. These misaligned primers can be extended by PolIV. Exhibits no 3'-5' exonuclease (proofreading) activity. May be involved in translesional synthesis, in conjunction with the beta clamp from PolIII. | 0.934 |