| node1 | node2 | node1 accession | node2 accession | node1 annotation | node2 annotation | score |
| BF93_03140 | BF93_18905 | BF93_03140 | BF93_18905 | Hypothetical protein; Derived by automated computational analysis using gene prediction method: GeneMarkS+. | Peptigoglycan-binding protein LysM; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.530 |
| BF93_18905 | BF93_03140 | BF93_18905 | BF93_03140 | Peptigoglycan-binding protein LysM; Derived by automated computational analysis using gene prediction method: Protein Homology. | Hypothetical protein; Derived by automated computational analysis using gene prediction method: GeneMarkS+. | 0.530 |
| BF93_18905 | guaA | BF93_18905 | BF93_13835 | Peptigoglycan-binding protein LysM; Derived by automated computational analysis using gene prediction method: Protein Homology. | GMP synthase; Catalyzes the synthesis of GMP from XMP. | 0.666 |
| BF93_18905 | guaB | BF93_18905 | BF93_13780 | Peptigoglycan-binding protein LysM; Derived by automated computational analysis using gene prediction method: Protein Homology. | Inosine-5-monophosphate dehydrogenase; Catalyzes the conversion of inosine 5'-phosphate (IMP) to xanthosine 5'-phosphate (XMP), the first committed and rate-limiting step in the de novo synthesis of guanine nucleotides, and therefore plays an important role in the regulation of cell growth. Belongs to the IMPDH/GMPR family. | 0.619 |
| BF93_18905 | lexA | BF93_18905 | BF93_18900 | Peptigoglycan-binding protein LysM; Derived by automated computational analysis using gene prediction method: Protein Homology. | ArsR family transcriptional regulator; Represses a number of genes involved in the response to DNA damage (SOS response), including recA and lexA. In the presence of single-stranded DNA, RecA interacts with LexA causing an autocatalytic cleavage which disrupts the DNA-binding part of LexA, leading to derepression of the SOS regulon and eventually DNA repair. | 0.553 |
| BF93_18905 | nrdR | BF93_18905 | BF93_18910 | Peptigoglycan-binding protein LysM; Derived by automated computational analysis using gene prediction method: Protein Homology. | NrdR family transcriptional regulator; Negatively regulates transcription of bacterial ribonucleotide reductase nrd genes and operons by binding to NrdR- boxes; Belongs to the NrdR family. | 0.718 |
| guaA | BF93_18905 | BF93_13835 | BF93_18905 | GMP synthase; Catalyzes the synthesis of GMP from XMP. | Peptigoglycan-binding protein LysM; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.666 |
| guaA | guaB | BF93_13835 | BF93_13780 | GMP synthase; Catalyzes the synthesis of GMP from XMP. | Inosine-5-monophosphate dehydrogenase; Catalyzes the conversion of inosine 5'-phosphate (IMP) to xanthosine 5'-phosphate (XMP), the first committed and rate-limiting step in the de novo synthesis of guanine nucleotides, and therefore plays an important role in the regulation of cell growth. Belongs to the IMPDH/GMPR family. | 0.999 |
| guaB | BF93_18905 | BF93_13780 | BF93_18905 | Inosine-5-monophosphate dehydrogenase; Catalyzes the conversion of inosine 5'-phosphate (IMP) to xanthosine 5'-phosphate (XMP), the first committed and rate-limiting step in the de novo synthesis of guanine nucleotides, and therefore plays an important role in the regulation of cell growth. Belongs to the IMPDH/GMPR family. | Peptigoglycan-binding protein LysM; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.619 |
| guaB | guaA | BF93_13780 | BF93_13835 | Inosine-5-monophosphate dehydrogenase; Catalyzes the conversion of inosine 5'-phosphate (IMP) to xanthosine 5'-phosphate (XMP), the first committed and rate-limiting step in the de novo synthesis of guanine nucleotides, and therefore plays an important role in the regulation of cell growth. Belongs to the IMPDH/GMPR family. | GMP synthase; Catalyzes the synthesis of GMP from XMP. | 0.999 |
| lexA | BF93_18905 | BF93_18900 | BF93_18905 | ArsR family transcriptional regulator; Represses a number of genes involved in the response to DNA damage (SOS response), including recA and lexA. In the presence of single-stranded DNA, RecA interacts with LexA causing an autocatalytic cleavage which disrupts the DNA-binding part of LexA, leading to derepression of the SOS regulon and eventually DNA repair. | Peptigoglycan-binding protein LysM; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.553 |
| lexA | nrdR | BF93_18900 | BF93_18910 | ArsR family transcriptional regulator; Represses a number of genes involved in the response to DNA damage (SOS response), including recA and lexA. In the presence of single-stranded DNA, RecA interacts with LexA causing an autocatalytic cleavage which disrupts the DNA-binding part of LexA, leading to derepression of the SOS regulon and eventually DNA repair. | NrdR family transcriptional regulator; Negatively regulates transcription of bacterial ribonucleotide reductase nrd genes and operons by binding to NrdR- boxes; Belongs to the NrdR family. | 0.640 |
| nrdR | BF93_18905 | BF93_18910 | BF93_18905 | NrdR family transcriptional regulator; Negatively regulates transcription of bacterial ribonucleotide reductase nrd genes and operons by binding to NrdR- boxes; Belongs to the NrdR family. | Peptigoglycan-binding protein LysM; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.718 |
| nrdR | lexA | BF93_18910 | BF93_18900 | NrdR family transcriptional regulator; Negatively regulates transcription of bacterial ribonucleotide reductase nrd genes and operons by binding to NrdR- boxes; Belongs to the NrdR family. | ArsR family transcriptional regulator; Represses a number of genes involved in the response to DNA damage (SOS response), including recA and lexA. In the presence of single-stranded DNA, RecA interacts with LexA causing an autocatalytic cleavage which disrupts the DNA-binding part of LexA, leading to derepression of the SOS regulon and eventually DNA repair. | 0.640 |