| node1 | node2 | node1 accession | node2 accession | node1 annotation | node2 annotation | score |
| OBX65744.1 | OBX65745.1 | A9309_01770 | A9309_01780 | Hypothetical protein; Derived by automated computational analysis using gene prediction method: GeneMarkS+. | Entericidin; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.421 |
| OBX65744.1 | dsrE | A9309_01770 | A9309_01765 | Hypothetical protein; Derived by automated computational analysis using gene prediction method: GeneMarkS+. | Multidrug transporter; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.960 |
| OBX65744.1 | glnE | A9309_01770 | A9309_01760 | Hypothetical protein; Derived by automated computational analysis using gene prediction method: GeneMarkS+. | Putative DNA modification/repair radical SAM protein; Involved in the regulation of glutamine synthetase GlnA, a key enzyme in the process to assimilate ammonia. When cellular nitrogen levels are high, the C-terminal adenylyl transferase (AT) inactivates GlnA by covalent transfer of an adenylyl group from ATP to specific tyrosine residue of GlnA, thus reducing its activity. Conversely, when nitrogen levels are low, the N-terminal adenylyl removase (AR) activates GlnA by removing the adenylyl group by phosphorolysis, increasing its activity. The regulatory region of GlnE binds the signa [...] | 0.540 |
| OBX65744.1 | tusE | A9309_01770 | A9309_01775 | Hypothetical protein; Derived by automated computational analysis using gene prediction method: GeneMarkS+. | Sulfite reductase; Part of a sulfur-relay system. | 0.701 |
| OBX65745.1 | OBX65744.1 | A9309_01780 | A9309_01770 | Entericidin; Derived by automated computational analysis using gene prediction method: Protein Homology. | Hypothetical protein; Derived by automated computational analysis using gene prediction method: GeneMarkS+. | 0.421 |
| OBX65745.1 | dsrE | A9309_01780 | A9309_01765 | Entericidin; Derived by automated computational analysis using gene prediction method: Protein Homology. | Multidrug transporter; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.421 |
| OBX65745.1 | tusE | A9309_01780 | A9309_01775 | Entericidin; Derived by automated computational analysis using gene prediction method: Protein Homology. | Sulfite reductase; Part of a sulfur-relay system. | 0.457 |
| dsrE | OBX65744.1 | A9309_01765 | A9309_01770 | Multidrug transporter; Derived by automated computational analysis using gene prediction method: Protein Homology. | Hypothetical protein; Derived by automated computational analysis using gene prediction method: GeneMarkS+. | 0.960 |
| dsrE | OBX65745.1 | A9309_01765 | A9309_01780 | Multidrug transporter; Derived by automated computational analysis using gene prediction method: Protein Homology. | Entericidin; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.421 |
| dsrE | glnE | A9309_01765 | A9309_01760 | Multidrug transporter; Derived by automated computational analysis using gene prediction method: Protein Homology. | Putative DNA modification/repair radical SAM protein; Involved in the regulation of glutamine synthetase GlnA, a key enzyme in the process to assimilate ammonia. When cellular nitrogen levels are high, the C-terminal adenylyl transferase (AT) inactivates GlnA by covalent transfer of an adenylyl group from ATP to specific tyrosine residue of GlnA, thus reducing its activity. Conversely, when nitrogen levels are low, the N-terminal adenylyl removase (AR) activates GlnA by removing the adenylyl group by phosphorolysis, increasing its activity. The regulatory region of GlnE binds the signa [...] | 0.540 |
| dsrE | tusE | A9309_01765 | A9309_01775 | Multidrug transporter; Derived by automated computational analysis using gene prediction method: Protein Homology. | Sulfite reductase; Part of a sulfur-relay system. | 0.995 |
| glnE | OBX65744.1 | A9309_01760 | A9309_01770 | Putative DNA modification/repair radical SAM protein; Involved in the regulation of glutamine synthetase GlnA, a key enzyme in the process to assimilate ammonia. When cellular nitrogen levels are high, the C-terminal adenylyl transferase (AT) inactivates GlnA by covalent transfer of an adenylyl group from ATP to specific tyrosine residue of GlnA, thus reducing its activity. Conversely, when nitrogen levels are low, the N-terminal adenylyl removase (AR) activates GlnA by removing the adenylyl group by phosphorolysis, increasing its activity. The regulatory region of GlnE binds the signa [...] | Hypothetical protein; Derived by automated computational analysis using gene prediction method: GeneMarkS+. | 0.540 |
| glnE | dsrE | A9309_01760 | A9309_01765 | Putative DNA modification/repair radical SAM protein; Involved in the regulation of glutamine synthetase GlnA, a key enzyme in the process to assimilate ammonia. When cellular nitrogen levels are high, the C-terminal adenylyl transferase (AT) inactivates GlnA by covalent transfer of an adenylyl group from ATP to specific tyrosine residue of GlnA, thus reducing its activity. Conversely, when nitrogen levels are low, the N-terminal adenylyl removase (AR) activates GlnA by removing the adenylyl group by phosphorolysis, increasing its activity. The regulatory region of GlnE binds the signa [...] | Multidrug transporter; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.540 |
| glnE | tusE | A9309_01760 | A9309_01775 | Putative DNA modification/repair radical SAM protein; Involved in the regulation of glutamine synthetase GlnA, a key enzyme in the process to assimilate ammonia. When cellular nitrogen levels are high, the C-terminal adenylyl transferase (AT) inactivates GlnA by covalent transfer of an adenylyl group from ATP to specific tyrosine residue of GlnA, thus reducing its activity. Conversely, when nitrogen levels are low, the N-terminal adenylyl removase (AR) activates GlnA by removing the adenylyl group by phosphorolysis, increasing its activity. The regulatory region of GlnE binds the signa [...] | Sulfite reductase; Part of a sulfur-relay system. | 0.494 |
| tusE | OBX65744.1 | A9309_01775 | A9309_01770 | Sulfite reductase; Part of a sulfur-relay system. | Hypothetical protein; Derived by automated computational analysis using gene prediction method: GeneMarkS+. | 0.701 |
| tusE | OBX65745.1 | A9309_01775 | A9309_01780 | Sulfite reductase; Part of a sulfur-relay system. | Entericidin; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.457 |
| tusE | dsrE | A9309_01775 | A9309_01765 | Sulfite reductase; Part of a sulfur-relay system. | Multidrug transporter; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.995 |
| tusE | glnE | A9309_01775 | A9309_01760 | Sulfite reductase; Part of a sulfur-relay system. | Putative DNA modification/repair radical SAM protein; Involved in the regulation of glutamine synthetase GlnA, a key enzyme in the process to assimilate ammonia. When cellular nitrogen levels are high, the C-terminal adenylyl transferase (AT) inactivates GlnA by covalent transfer of an adenylyl group from ATP to specific tyrosine residue of GlnA, thus reducing its activity. Conversely, when nitrogen levels are low, the N-terminal adenylyl removase (AR) activates GlnA by removing the adenylyl group by phosphorolysis, increasing its activity. The regulatory region of GlnE binds the signa [...] | 0.494 |