| node1 | node2 | node1 accession | node2 accession | node1 annotation | node2 annotation | score |
| ANP63875.1 | ANP64252.1 | BAU10_02280 | BAU10_04370 | DNA repair protein RecN; May be involved in recombinational repair of damaged DNA. | UV protection and mutation protein; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.671 |
| ANP63875.1 | ANP64481.1 | BAU10_02280 | BAU10_05610 | DNA repair protein RecN; May be involved in recombinational repair of damaged DNA. | Hypothetical protein; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | 0.669 |
| ANP63875.1 | ANP64635.1 | BAU10_02280 | BAU10_06420 | DNA repair protein RecN; May be involved in recombinational repair of damaged DNA. | DNA polymerase V; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.671 |
| ANP63875.1 | ANP65494.1 | BAU10_02280 | BAU10_10985 | DNA repair protein RecN; May be involved in recombinational repair of damaged DNA. | Multidrug DMT transporter permease; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.738 |
| ANP63875.1 | ANP65587.1 | BAU10_02280 | BAU10_11480 | DNA repair protein RecN; May be involved in recombinational repair of damaged DNA. | UV protection and mutation protein; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.671 |
| ANP63875.1 | ANP65638.1 | BAU10_02280 | BAU10_11760 | DNA repair protein RecN; May be involved in recombinational repair of damaged DNA. | DNA polymerase V; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.671 |
| ANP63875.1 | lexA | BAU10_02280 | BAU10_14410 | DNA repair protein RecN; May be involved in recombinational repair of damaged DNA. | Repressor LexA; Represses a number of genes involved in the response to DNA damage (SOS response), including recA and lexA. In the presence of single-stranded DNA, RecA interacts with LexA causing an autocatalytic cleavage which disrupts the DNA-binding part of LexA, leading to derepression of the SOS regulon and eventually DNA repair. | 0.797 |
| ANP63875.1 | recA | BAU10_02280 | BAU10_12345 | DNA repair protein RecN; May be involved in recombinational repair of damaged DNA. | Recombinase RecA; Can catalyze the hydrolysis of ATP in the presence of single- stranded DNA, the ATP-dependent uptake of single-stranded DNA by duplex DNA, and the ATP-dependent hybridization of homologous single-stranded DNAs. It interacts with LexA causing its activation and leading to its autocatalytic cleavage; Belongs to the RecA family. | 0.930 |
| ANP64252.1 | ANP63875.1 | BAU10_04370 | BAU10_02280 | UV protection and mutation protein; Derived by automated computational analysis using gene prediction method: Protein Homology. | DNA repair protein RecN; May be involved in recombinational repair of damaged DNA. | 0.671 |
| ANP64252.1 | ANP64481.1 | BAU10_04370 | BAU10_05610 | UV protection and mutation protein; Derived by automated computational analysis using gene prediction method: Protein Homology. | Hypothetical protein; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | 0.550 |
| ANP64252.1 | ANP65494.1 | BAU10_04370 | BAU10_10985 | UV protection and mutation protein; Derived by automated computational analysis using gene prediction method: Protein Homology. | Multidrug DMT transporter permease; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.662 |
| ANP64252.1 | recA | BAU10_04370 | BAU10_12345 | UV protection and mutation protein; Derived by automated computational analysis using gene prediction method: Protein Homology. | Recombinase RecA; Can catalyze the hydrolysis of ATP in the presence of single- stranded DNA, the ATP-dependent uptake of single-stranded DNA by duplex DNA, and the ATP-dependent hybridization of homologous single-stranded DNAs. It interacts with LexA causing its activation and leading to its autocatalytic cleavage; Belongs to the RecA family. | 0.901 |
| ANP64481.1 | ANP63875.1 | BAU10_05610 | BAU10_02280 | Hypothetical protein; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | DNA repair protein RecN; May be involved in recombinational repair of damaged DNA. | 0.669 |
| ANP64481.1 | ANP64252.1 | BAU10_05610 | BAU10_04370 | Hypothetical protein; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | UV protection and mutation protein; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.550 |
| ANP64481.1 | ANP64635.1 | BAU10_05610 | BAU10_06420 | Hypothetical protein; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | DNA polymerase V; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.550 |
| ANP64481.1 | ANP65494.1 | BAU10_05610 | BAU10_10985 | Hypothetical protein; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | Multidrug DMT transporter permease; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.844 |
| ANP64481.1 | ANP65587.1 | BAU10_05610 | BAU10_11480 | Hypothetical protein; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | UV protection and mutation protein; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.550 |
| ANP64481.1 | ANP65638.1 | BAU10_05610 | BAU10_11760 | Hypothetical protein; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | DNA polymerase V; Derived by automated computational analysis using gene prediction method: Protein Homology. | 0.550 |
| ANP64481.1 | lexA | BAU10_05610 | BAU10_14410 | Hypothetical protein; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | Repressor LexA; Represses a number of genes involved in the response to DNA damage (SOS response), including recA and lexA. In the presence of single-stranded DNA, RecA interacts with LexA causing an autocatalytic cleavage which disrupts the DNA-binding part of LexA, leading to derepression of the SOS regulon and eventually DNA repair. | 0.670 |
| ANP64481.1 | recA | BAU10_05610 | BAU10_12345 | Hypothetical protein; Component of the SOS system and an inhibitor of cell division. Accumulation of SulA causes rapid cessation of cell division and the appearance of long, non-septate filaments. In the presence of GTP, binds a polymerization-competent form of FtsZ in a 1:1 ratio, thus inhibiting FtsZ polymerization and therefore preventing it from participating in the assembly of the Z ring. This mechanism prevents the premature segregation of damaged DNA to daughter cells during cell division. | Recombinase RecA; Can catalyze the hydrolysis of ATP in the presence of single- stranded DNA, the ATP-dependent uptake of single-stranded DNA by duplex DNA, and the ATP-dependent hybridization of homologous single-stranded DNAs. It interacts with LexA causing its activation and leading to its autocatalytic cleavage; Belongs to the RecA family. | 0.636 |